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In a series of 162 men with metastatic prostate cancer, a multivariate approach defined the best prognostic model for survival based on 6-month testosterone levels of men treated with goserelin.

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There are two types of brachytherapy, both of which are done under general anesthesia.

Read my story on YANA prostate cancer survivor stories by the name Zorro.

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This difference in effectiveness in men and women may be related to the fact that NSAAs like flutamide significantly increase androgen levels in men, which counteracts their antiandrogen efficacy, but do not increase androgen levels in women.

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Deforolimus in combination with MK-2206 is highly effective for inhibiting castration-resistant prostate cancer (CRPC) in preclinical studies in vivo using a refined genetically engineered mouse model of the disease.

For those cells, you need hormone therapy.

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Of 30 patients, 33% had organ-confined disease, 70% had extraprostatic extension, 37% had positive lymph nodes, and 17% exhibited positive surgical margins.

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Activities should be restricted for the first 24 hours after biopsy.

These medications, however, may increase androgen secretion by blocking or reducing the negative feedback of estrogen on the hypothalamic-pituitary axis, so caution should be exercised in patients with prostate cancer until more safety and efficacy data are available.

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While the well-understood products of the adrenal gland are mineralocorticoids (aldosterone, corticosterone), glucocorticoids (cortisol), and the adrenal androgens (DHEA-S, AED and small amounts of T), the activity of several other intermediates with potential relevance to CRPC have recently been elucidated.

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ADT consists of treatment with a GnRH agonist, combined androgen blockade (GnRH analog plus antiandrogen), or, less commonly, bilateral orchiectomy.

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Initial hormonal management of androgen-sensitive metastatic, recurrent, or progressive prostate cancer: 2006 update of an American Society of Clinical Oncology practice guideline.

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Food and Drug Administration for the treatment of patients with metastatic castration-resistant prostate cancer (CRPC).

His research expertise is on targeted therapies for castration-resistant prostate cancer, using kinase inhibitors, androgen receptor signalling inhibitors, and autophagy modulators.

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Intratumoral de novo steroid synthesis activates androgen receptor in castration-resistant prostate cancer and is upregulated by treatment with CYP17A1 inhibitors.

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CASODEX (bicalutamide) orodispersible tablet was studied in combination with ARIMIDEX (anastrozole) orodispersible tablet in an open-label, non-comparative, multi-center study that assessed the efficacy and safety of this combination regimen over 12 months in the treatment of gonadotropin-independent precocious puberty in boys with familial male-limited precocious puberty, also known as testotoxicosis.

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The Panel did not include the other treatment options in the analysis and recommendations due to a combination of factors, including limited published experience and short-term follow-up as well as the similar issues that affected evaluations of other treatment options (see the "Methodology" and the "Summary of Treatment Complications" sections for an explanation of data limitations).

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The Panel feels that because of the substantial differences among disease stage, especially between clinical stage T1 to T2 and T3 to T4 disease, any future studies including both groups of subjects should report all data stratified by T1 to T2.

Dad received four injections and last month we did surgery and removed his testicals, now doctors has stopped hormone therapy.

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Abstracts were screened for articles reporting outcomes (efficacy and safety) of prostate cancer treatment in patients with clinical stage T1 or T2 disease.

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If the cancer continues to grow, chemotherapy or immunotherapy may be recommended.

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Impact of Prior Use of an Androgen Receptor-Axis-Targeted (ARAT) Agent With or Without Subsequent Taxane Therapy on the Efficacy of Another ARAT Agent in Patients With Metastatic Castration-Resistant Prostate Cancer.

Strategies to block androgen-receptor signaling have formed the backbone of prostate-cancer therapy since the first description of the hormonal dependence of this cancer in 1941.

The disease is either classified as acute or chronic, referring to how quickly it develops and progresses.

Some studies have demonstrated equivalence or a slight advantage for intermittent therapy; however, until larger clinical trials have been completed, intermittent hormonal therapy is still considered somewhat experimental.

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